"Doc, I'm just getting old." This is a common excuse we hear
from runners seeking to explain an injury or poor performance.
So common is this excuse that we have given it a name: Old Age
Syndrome (OAS).OAS is not a physical affliction but rather a state of mind.
This mind set allows us to disregard the true causes of injury
and have less guilt about the decline in our performance. If
you suffer from OAS, do not despair. The treatment is simple:
understand the true causes of injuries and examine the
physiological research out of Ball State University on aging
runners.
Running is a bilateral sport where equal demand is placed on
both sides of the body.
However, most running injuries occur on only one side. This is
due to muscle inflexibility and strength imbalances, along with
faulty skeletal alignment, causing altered biomechanical
function.
Altered biomechanics in turn focus stress unequally on various
parts of the body. These areas become the subject of overuse
and are ultimately the sites of injury.
Injuries are not a function of age. It is amazing how many
doctors use the infamous line "you are not getting any
younger," when you describe your latest affliction. Aging only
allows more time for our insufficiencies to catch up with us,
rather than actually being a cause. Next time you injure a leg;
remind yourself that the other leg is the same age.
Runners with OAS will carry the same injuries with them for
years like a badge of courage, as they limp through workouts
and races, never taking steps to actually deal with the cause
of the injury, or seek treatment and rehabilitation advice.
Various endurance factors including heart rate, VO2 max and
muscle structure were measured at Ball State University on a
group of elite runners in their late 40s and compared to the
values they had when tested in their late 20s.
Physiologically, running is controlled by three major factors:
the heart (the pump), lungs (oxygen supply), and the muscles
(our wheels). If aging drastically affects our running, it
should certainly be reflected by one of these three factors.
If Old Age Syndrome causes a decrease in performance over time,
surely it must affect the capacity of the heart to do endurance
work over time. Yet, when the runners were tested at age 48,
maximal heart rates and sub maximal heart rates (rate required
to maintain a particular pace) were "well maintained" when
compared to that at age 26. The heart's capacity to handle
endurance running did not decline over the years.
Then OAS must affect the lungs and oxygen transport to decrease
performance. Yet, the runners had no significant loss of VO2
max between ages 26 and 48. As a matter of fact, an additional
study showed maintenance of VO2 max between the ages of 52 and
62. The runners also maintained their running efficiency (the
oxygen cost of maintaining a running pace) over the decades.
Their bodies were not working harder to run the same pace,
neither when measured by the heart nor the lungs.
If OAS does not diminish heart and lung capacity, then the
muscles must be the limiting factor, as structure and strength
changes occur over time, right? Think again. The runners had
consistent running weight from 26 to 48 with little deviation,
some actually weighed slightly less.
And rather than the muscle cells shrinking over time (a popular
belief), the runners had a twenty-five percent increase in the
cross-sectional size of endurance muscle cells (Type 1) from
age 26 to 48. Blood supply to the muscle cells was also
similar, meaning oxygen transport to the cells and waste
product removal can occur as efficiently in the older runners.
The muscles were shown to be as capable, if not more so, to
carry out endurance activity.
Some may be thinking at this point: "they may have the
endurance, but stiffness and decreasing flexibility must change
their stride length and slow them down." Fact: The stride
length of the runners remained the same, and had not decreased
at all over the years. This good news is not limited to those
in their 40s. Other studies have shown strength training in 60
to 72 year old men led to increased blood supply to their
muscles by fifteen percent. Regular exercise regimens
instituted by previously inactive 70 to 79 year olds increased
their VO2 max by twenty to twenty-five percent, indicating
greater endurance capacities for older sedentary individuals if
they exercise.
This research, carried out on elite runners who continued
running over the years, shows the potential of the human body
to maintain endurance fitness levels over the decades before
serious decline is noted. One needs only to look at the
accomplishments of Eamonn Coughlan
(3:58 mile), Doug Kurtis (2:15 Boston Marathon), and Priscilla
Welch (New York Marathon victory as a master) for inspiration
to what can be accomplished. Locally, Chuck Moeser, Alisa
Harvey, and Jim Hage continually trounce the majority of
younger runners in the area. Declining performances and
increasing times through the 40s and beyond have more to do
with less consistent training at quality speeds and by
increasing family and work responsibilities which we did not
have to endure in our twenty-something years. Priorities shift
and motivation wanes as goals become readjusted with the
various stresses of life. Injuries seem a little harder to
bounce back from, which also affects the long-term consistency
of training.
There is a point where aging and performance intersect and
continue on diverging paths.
The importance of this research is that those in their 40s,
50s, and beyond can reset their goals and expect better
performances because the body still has the capacity to improve
if training is consistently followed and injuries are avoided.
Everyone needs to assess their priorities and balance their
training with the other responsibilities of life. In reality,
Old Age Syndrome does not have to dampen your goals over the
years. For most of us who continue to run vigorously over the
years, it just doesn't exist! Free your mind!